Eating less of this one nutrient may “switch off” cancer growth, research suggests
For years, what we believed and understood about cancer came from one basic thought. We assumed that cancer begins with an environmental chemical carcinogen or some radiation that causes mutation in a normal cell to a cancer-prone cell. These cells then grow into a cluster of cells through a series of mutations, ultimately giving us the cells that become diagnosed as cancer.
That is the mutation theory of cancer. To avoid cancer means avoiding these changes, like not smoking and avoiding too much direct sunlight. Failing this, we have to selectively kill cancer cells through treatment like surgery, chemotherapy, or radiation. We don’t expect mutated cells to become healthy again because the mutation is not reversible.
A recent report relied on this theory of mutation to explain why some of our tissues have higher rates of cancer. They found that high rate cancer tissues have a higher rate of cell division. They also discovered that only one-third of these mutations are caused by lifestyle or environmental factors. The other two-thirds of cancers were said to be random, ‘bad luck’ cancers.
It concluded that whether random or purposeful, both types of cancer rely on this mutation theory of cancer, which serves as a fundamental rationale for the cancer industry of research, development, and clinical practice. But there is evidence that cancer may not just be bad luck, but predicated on something tangible.
Cancer and Diet
A study performed by Dr. T. Colin Campbell investigated in laboratory rats how cancer, once initiated by mutation, is not further developed by a series of additional mutations.
They found that cancer was initiated by a gene mutation caused by a powerful chemical carcinogen called aflatoxin. But they also discovered that cancer development was not caused by additional mutations.
They found that cancer development was predominantly promoted by feeding protein levels that exceeded the amount required for good health, but still typical of most human diets. Cancer development couldn’t be caused by additional mutations because the original mutation-producing chemical was no longer present during the promotion.
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This suggested that, although cells are primed for cancer development by mutation, further progression to a cancer diagnosis is nutritionally controlled by non-mutagenic mechanisms.
When protein consumption was reduced to levels that were conducive to good health, cancer growth was reversed. They found that cancer growth could be turned off, and on, and off again, by a nutritional protocol that did not involve mutation. They also discovered that cancer growth occurred with animal-based protein and not plant-based.
Animal-based protein was found to increase a growth hormone that encourages cancer growth, compromises the body’s cells that normally destroy cancer cells and minimizes calories being used for cancer growth.
Initial mutations that prime cells for cancer may live dormant for years in a body on a low-protein diet, but can be activated to support new cancer growth when a high animal-protein diet is restored.
Where’s The Evidence?
There is human evidence that supports this claim; countries can have high levels of certain cancers, and others may have levels closer to zero. The difference being diet and lifestyle. Other studies showed that people are migrating from one country to another, in a generation or so, assumed the cancer rates of their host country with no changes to their genes, only their dietary habits.
This theory is highly disregarded by many researchers because they do not understand the power of nutrition and cling to the original theory of cancer mutation. This is allowing for more funding and research into different drugs that come with side effects that are almost as bad as the disease they are claiming to cure.
It is time for people to understand that cancer isn’t just random and that it isn’t just based on mutation, it’s also based on nutrition.
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